In his latest column, David Howett reviews a recent paper from Gunn-Moore et al. on the relationship between Alzheimer’s disease and Type 2 diabetes.
New study demonstrates how the enzyme, ACSS2, contributes to brain tumor growth in environments scarce of oxygen and nutrients.
Researchers have developed computer models to further understanding the behavior of protein fragments and fibril aggregation in Huntington’s disease development.
In this Editorial, researchers from McGovern Medical School put the recent Lennox et al. Lancet paper in context, looking at the role of neuronal cell surface antibodies in schizophrenia.
The study from researchers at Michigan State University (MI, USA) also demonstrates that an antihistamine is effective in preventing prion aggregation.
Accumulating evidence has implicated a role for aquaporins in the nervous system, including reports of AQP1 and AQP4 in pain and neuroinflammation.
Researchers have identified a dysfunctional cell receptor in the immune system that could be part of the cause of chronic fatigue syndrome.
A recent study has identified a cellular pathway that that may lead to the protein misfolding observed in those diagnosed with Huntington’s disease.
A new mechanism that controls nerve cell communication has been identified, which could be a major advance in understanding neurodegenerative disorders.