A team of neuroscientists has identified a group of cells in the hippocampus that can control fear memories. Understanding the mechanisms by which these cells activate and suppress memories could improve therapies for fear-based disorders.
Novel study suggests REM sleep dampens the brain’s sensitivity to fearful stimuli.
According to new research, the antibiotic disrupts creation of fear memories in the brain, which could be utilized for treatment or prevention of post-traumatic stress disorder.
Researchers have uncovered a role for the gene FKBP5 in memory formation following traumatic events, and suggest that manipulation of the gene could help prevent PTSD.
Biological underpinnings of trauma and post-traumatic stress disorder: focusing on genetics and epigenetics
The authors highlight some of the key genetic risk factors identified for PTSD, and the evidence that epigenetic processes might play a role in the biological response to trauma, as well as being potential biomarkers of PTSD risk. We also discuss important considerations for future research in this area.
Chinese researchers have utilized MRI to demonstrate that children with PTSD experience disruptions in the structure of their neural networks.
Researchers identify a distinct group of hypothalamic neurons implicated in anxiety that could be targeted in future treatments.
Hcn1, a gene that regulates neuronal excitability, has been identified in mice as a potential target for the treatment of trauma- and stress-related disorders, and may explain symptom and treatment response variability.