Excessive stress during childhood or fetal development can have long-term consequences on the brain, with research from SfN Neuroscience uncovering new mechanisms and therapeutic targets.
A series of research presented at SfN’s Neuroscience 2017 has shed more light on the mechanism by which stress negatively impacts the brain, potentially highlighting future therapeutics.
Researchers from Japan have revealed a new neural pathway that links stress with organ dysfunction.
Webinar Q&A follow-up: How chronic stress experienced during early development epigenetically programs adult disease risk
James Coffman responds to questions from our recent webinar, ‘How chronic stress experienced during early development epigenetically programs adult disease risk’.
According to new research, the antibiotic disrupts creation of fear memories in the brain, which could be utilized for treatment or prevention of post-traumatic stress disorder.
Authors also claim that stress-reduction techniques could be an effective low-risk treatment for sufferers.
Dr Coffman is currently studying the mechanisms by which chronic early-life stress increases adult disease risk, using zebrafish as a model organism to ask how such stress affects immune system development and regulation.
Epidemiological studies have shown that chronic stress experienced very early in life—even prenatally—increases the risk of developing inflammatory disease in adulthood, including mental health disorders such as anxiety and depression.
Veronika Kiryanova discusses her recent work on maternal stress and offspring mental health, and her thoughts on how research such as this could impact future research and clinical practice.
A review of biological pathways and mechanisms underlying the social gradient in health, with particular emphasis on behavioral stress influences.